The distinguished authors of The Dyslexia Debate have mistakenly overlooked challenging research proving dyslexia’s existence and solving its many riddles.
- What if dyslexics do not have primary cerebral problems in decoding or comprehending the clear reading signals received as traditionally believed for over a century?
- What if dyslexia is a diverse cluster of coexisting reading and non-reading symptoms varying from severe to overcompensated rather than just a major reading comprehension disorder?
- What if the invariable presence of only balance and coordination neurological signs diagnostic of an inner-ear and cerebellar dysfunction among all dyslexics were evidence that dyslexia is of a primary cerebellar origin, and that their thinking brains were normal and even superior?
- What if all dyslexic symptoms were caused by normal brains failing to secondarily process the inner-ear determined scrambled or “dizzy” signals received and transmitted?
- What if “transient dyslexic” reading and non-reading symptoms normally arise after spinning and disappear when dizzy sensations and signals improve spontaneously and/or following treatment with inner-ear-improving medications and related therapies?
About Harold Levinson, M.D.
Formerly Clinical Associate Professor of Psychiatry at New York University Medical Center, Dr. Harold Levinson is currently Director of the Levinson Medical Center for Learning Disabilities in Long Island, New York. He is a well-known neuropsychiatrist, clinical researcher and author.
By Harold Levinson, M.D.